Bearded dragon hazards — metabolic bone disease, vitamin A deficiency, and UVB dependence
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Secondary nutritional hyperparathyroidism is the most common bone disease encountered in reptile practice.It is caused by poor diet (low calcium:phosphorus ratio, vitamin D3 deficiency) or poor husbandry (lack of UVB light, inadequate thermal provision)UVB light (290–300 nm) is especially important for most diurnal lizards and chelonians for vitamin D3 synthesis and calcium regulation.Hypovitaminosis A is common in aquatic turtles and insectivorous lizards fed a diet deficient in preformed vitamin A.The commonly used nutritional supplement beta carotene cannot be metabolized by many reptiles.However, UVB light (290–300 nm) is especially important for most diurnal lizards and chelonians for vitamin D3 synthesis and calcium regulation.Species identification is essential to critically appraise captive diets (see the tables Composition of Animal Foods That May Be Offered to Reptiles and Composition of Plant Foods That May Be Offered to Reptiles).It is caused by poor diet (low calcium:phosphorus ratio, vitamin D3 deficiency) or poor husbandry (lack of UVB light, inadequate thermal provision) (see secondary nutritional hyperparathyroidism photograph and radiograph).Traditionally, Paget disease has been characterized by repeated episodes of osteoclastic bone resorption and deposition leading to dense, brittle bones.
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Source: pdf-raw/exotic-toxicology/reptile.txt · https://www.merckvetmanual.com/